2019-nCov Coronavirus
Posted: Tue Feb 04, 2020 11:16 am
What's out there about the Coronavirus...
There are seven kinds of Coronavirus that can infect humans.
There are four known as Common Coronavirus which people around the world commonly get infected with.
They are four sub groups Alpha, Beta, Gamma and Delta...
229E (Alpha Coronavirus)
NL63 (Alpha Coronairus)
OC43 (Beta Coronavirus)
HKU1 (Beta Coronavirus)
2002-SARS-Cov (Beta Coronavirus) Severe Acute Respiratory Syndrome
2012-MERS-Cov (Beta Coronavirus) Middle Eastern Respiratory Syndrome
2019-nCov (Beta Coronavirus) Novel Coronavirus
SARS, MERS and nCov are mutation viruses from animals
Coronavirus is an Airborne virus, spreads similar to colds & flu - small droplets of saliva coughed or sneezed into the air, also spread indirectly via surfaces like door handles.
SARS Kill Rate 1 IN 10 (8'098 Cases with 774 Deaths) incubation period of 2 to 7 Days - Source UK National Health Service NHS website
MERS Kill Rate 1 IN 3 (2'494 Cases with 858 Deaths) 34% mortality rate with an incubation period of 4 to 8 Days - Source World Health Organisation WHO
nCoV Kill Rate for now they say 1 IN 30 but data coming out each day suggests a higher mortality rate.
Like it's siblings SARS and MERS, the new nCov virus causes Pneumonia
How Did The Novel Coronavirus Epidemic Start?
The Novel Coronavirus (2019-nCoV) is a new strain of Coronavirus that has caused a pneumonia outbreak in China. Experts suspected that the virus is transmitted from animals to humans based on the first case found in the city of Wuhan.
However, there are mixed reports on when and how the virus started. A recent Wuhan City Administration and the World Health Organization (WHO) report stated that the first individuals were infected between December 12 to December 29 while working at an unsanitary seafood market in Wuhan which was later shut down. Subsequently to the report, however, a scientific paper written by a group of Chinese researchers published in The Lancet rebutted the previous claim by arguing that the first 13 out of 41 patients affected by the virus believed to be the 2019-nCoV had no contacts with the seafood market at all in early December.
“No epidemiological link was found between the first patient and later cases,” the report noted.
Daniel Lucey, an infectious disease specialist at Georgetown University, also questioned the accuracy of the previous report pinning the blame on the seafood market. He argued that if the WHO and Wuhan administration data were accurate, the first infection must have been in November 2019 because of the incubation period between infection and the apparent symptoms. If so, there would be human-to-human transmission occurring in Wuhan.
Currently there is NO CURE for SARS
Currently there is NO CURE for MERS
The incubation period for nCoV is much longer (probably 14 Days after infection)
and the enclosed pdf would suggest that the 2019-nCov virus has 4 additional HIV-1 Insertions that are not in any of the previous versions of Coranavirus.
Johns Hopkins University has a live Operational Dashboard giving out up to date info on 2019-nCov Global Cases.
https://gisanddata.maps.arcgis.com/apps ... 7b48e9ecf6
1st Feb 2020 Total Cases were 12'373 and 2nd Feb 2020 Total Cases were 14'637 (100/12373*14637-100) = 18.3% growth rate
1st Feb 2020 Total Deaths were 257 and 2nd Feb 2020 Total Deaths were 305 (100/257*305-100) = 18.6% growth rate
3rd Feb 2020 Total Cases were 17'318 (100/14'637*17'318-100) = 18.3% growth rate
3rd Feb 2020 Total Deaths were 362 (100/305*362-100) = 18.3% growth rate
4th Feb 2020 Total Cases were 20'659 (100/17'318*20'659-100) = 19.29% growth rate
4th Feb 2020 Total Deaths were 427 (100/362*427-100) = 17.95% growth rate
5th Feb 2020 Total Cases were 24'597 (100/20'659*24'597-100) = 19.06% growth rate
5th Feb 2020 Total Deaths were 494 (100/427*494-100) = 15.69% growth rate
6th Feb 2020 Total Cases were 28'078 (100/24'957*28'078-100) = 12.50% growth rate
6th Feb 2020 Total Deaths were 564 (100/494*564-100) = 14.17% growth rate
28'078 cases divided by 564 deaths would suggest 1 IN 49.78 similar to Spanish Flu 1 IN 50
Still not showing signs of letting up - still increasing exponentially in China
It is still too early, and available information is still too incomplete, to be certain about many aspects of the 2019 novel coronavirus (2019-nCoV) outbreak in China. However, important elements of the management of this global health emergency—and it is an emergency of major international concern—are becoming clearer. As the outbreak accelerates, there are early lessons to be learned. The transmissibility of 2019-nCoV—or at least its geographical distribution—seems to be higher and broader than initially expected. Why? Partly this may be because of China's rapid expansion of its transport networks, especially air and high-speed rail. Wuhan is a crucial hub: linking west to Chengdu, south to Guangzhou and Shenzhen, east to Nanjing and Shanghai, and north to Beijing. With much of December a period when the outbreak went unreported and unrecognised, the population exposed to the virus is far greater than first thought—a cause for heightened concern.
“No bats were sold or found at the Huanan seafood market”
Dr. Yuhong writes about The Lancet study by authors Roujian Lu et al., from the China Key Laboratory of Biosafety, National Institute for Viral Disease Control and Prevention, Chinese Center for Disease Control and Prevention, repeating a quote from that paper:
First, the outbreak was first reported in late December 2019, when most bat species in Wuhan are hibernating. Second, no bats were sold or found at the Huanan seafood market, whereas various non-aquatic animals (including mammals) were available for purchase. Third, the sequence identity between 2019-nCoV and its close relatives bat-SL-CoVZC45 and bat-SL-CoVZXC21 was less than 90%. Hence, bat-SL-CoVZC45 and bat-SL-CoVZXC21 are not direct ancestors of 2019-nCoV.
In other words, it isn’t from bats.
That means the entire mainstream media is lying to us about the real origins of the coronavirus.
That same paper goes on to underscore the misinformation in the official explanation, stating, “Many of the initially confirmed 2019-nCoV cases—27 of the first 41 in one report, 26 of 47 in another—were connected to the Wuhan market, but up to 45%, including the earliest handful, were not. This raises the possibility that the initial jump into people happened elsewhere.”
Both Lu (in The Lancet paper linked above) and Lyons-Weiler point to the presence of a SARS binding protein sequence in the coronavirus that allows it to easily infect human cells. As explained in The Epoch Times:
…despite considerable genetics distance between the Wuhan CoV and the human-infecting SARS-CoV, and the overall low homology of the Wuhan CoV S-protein to that of SARS-CoV, the Wuhan CoV S-protein had several patches of sequences in the receptor binding (RBD) domain with a high homology to that of SARS-CoV. The residues at positions 442, 472, 479, 487, and 491 in SARS-CoV S-protein were reported to be at receptor complex interface and considered critical for cross species and human-to-human transmission of SARS-CoV. So to our surprise, despite replacing four out of five important interface amino acid residues, the Wuhan CoV S-protein was found to have a significant binding affinity to human ACE2. …The Wuhan CoV S-protein and SARS-CoV S-protein shared an almost identical 3-D structure in the RBD domain, thus maintaining similar van der Waals and electrostatic properties in the interaction interface. Thus the Wuhan CoV is still able to pose a significant public health risk for human transmission via the S protein–ACE2 binding pathway. (emphasis added)
As Dr. Yuhong asks, “How could this novel virus be so intelligent as to mutate precisely at selected sites while preserving its binding affinity to the human ACE2 receptor? How did the virus change just four amino acids of the S-protein? Did the virus know how to use Clustered Regularly Interspaced Short Palindromic Repeats (CRISPR) to make sure this would happen?”
It couldn’t happen by chance, in other words. The coronavirus is not a random mutation in the wild. It was engineered.
Many other scientists around the world are now investigating the gene sequences found in the coronavirus, and they are increasingly concluding that elements of the virus have been engineered.
Many of those scientists are being threatened and censored. One paper has so far been forced to be withdrawn and revised, no doubt to remove the key conclusions that point to the genetic engineering origins of the coronavirus, but the proof of its engineering cannot be denied forever.
Hope this info is of some use to anyone.
If any of the above Data is wrong, please advise
All the best
Xard777
There are seven kinds of Coronavirus that can infect humans.
There are four known as Common Coronavirus which people around the world commonly get infected with.
They are four sub groups Alpha, Beta, Gamma and Delta...
229E (Alpha Coronavirus)
NL63 (Alpha Coronairus)
OC43 (Beta Coronavirus)
HKU1 (Beta Coronavirus)
2002-SARS-Cov (Beta Coronavirus) Severe Acute Respiratory Syndrome
2012-MERS-Cov (Beta Coronavirus) Middle Eastern Respiratory Syndrome
2019-nCov (Beta Coronavirus) Novel Coronavirus
SARS, MERS and nCov are mutation viruses from animals
Coronavirus is an Airborne virus, spreads similar to colds & flu - small droplets of saliva coughed or sneezed into the air, also spread indirectly via surfaces like door handles.
SARS Kill Rate 1 IN 10 (8'098 Cases with 774 Deaths) incubation period of 2 to 7 Days - Source UK National Health Service NHS website
MERS Kill Rate 1 IN 3 (2'494 Cases with 858 Deaths) 34% mortality rate with an incubation period of 4 to 8 Days - Source World Health Organisation WHO
nCoV Kill Rate for now they say 1 IN 30 but data coming out each day suggests a higher mortality rate.
Like it's siblings SARS and MERS, the new nCov virus causes Pneumonia
How Did The Novel Coronavirus Epidemic Start?
The Novel Coronavirus (2019-nCoV) is a new strain of Coronavirus that has caused a pneumonia outbreak in China. Experts suspected that the virus is transmitted from animals to humans based on the first case found in the city of Wuhan.
However, there are mixed reports on when and how the virus started. A recent Wuhan City Administration and the World Health Organization (WHO) report stated that the first individuals were infected between December 12 to December 29 while working at an unsanitary seafood market in Wuhan which was later shut down. Subsequently to the report, however, a scientific paper written by a group of Chinese researchers published in The Lancet rebutted the previous claim by arguing that the first 13 out of 41 patients affected by the virus believed to be the 2019-nCoV had no contacts with the seafood market at all in early December.
“No epidemiological link was found between the first patient and later cases,” the report noted.
Daniel Lucey, an infectious disease specialist at Georgetown University, also questioned the accuracy of the previous report pinning the blame on the seafood market. He argued that if the WHO and Wuhan administration data were accurate, the first infection must have been in November 2019 because of the incubation period between infection and the apparent symptoms. If so, there would be human-to-human transmission occurring in Wuhan.
Currently there is NO CURE for SARS
Currently there is NO CURE for MERS
The incubation period for nCoV is much longer (probably 14 Days after infection)
and the enclosed pdf would suggest that the 2019-nCov virus has 4 additional HIV-1 Insertions that are not in any of the previous versions of Coranavirus.
Johns Hopkins University has a live Operational Dashboard giving out up to date info on 2019-nCov Global Cases.
https://gisanddata.maps.arcgis.com/apps ... 7b48e9ecf6
1st Feb 2020 Total Cases were 12'373 and 2nd Feb 2020 Total Cases were 14'637 (100/12373*14637-100) = 18.3% growth rate
1st Feb 2020 Total Deaths were 257 and 2nd Feb 2020 Total Deaths were 305 (100/257*305-100) = 18.6% growth rate
3rd Feb 2020 Total Cases were 17'318 (100/14'637*17'318-100) = 18.3% growth rate
3rd Feb 2020 Total Deaths were 362 (100/305*362-100) = 18.3% growth rate
4th Feb 2020 Total Cases were 20'659 (100/17'318*20'659-100) = 19.29% growth rate
4th Feb 2020 Total Deaths were 427 (100/362*427-100) = 17.95% growth rate
5th Feb 2020 Total Cases were 24'597 (100/20'659*24'597-100) = 19.06% growth rate
5th Feb 2020 Total Deaths were 494 (100/427*494-100) = 15.69% growth rate
6th Feb 2020 Total Cases were 28'078 (100/24'957*28'078-100) = 12.50% growth rate
6th Feb 2020 Total Deaths were 564 (100/494*564-100) = 14.17% growth rate
28'078 cases divided by 564 deaths would suggest 1 IN 49.78 similar to Spanish Flu 1 IN 50
Still not showing signs of letting up - still increasing exponentially in China
It is still too early, and available information is still too incomplete, to be certain about many aspects of the 2019 novel coronavirus (2019-nCoV) outbreak in China. However, important elements of the management of this global health emergency—and it is an emergency of major international concern—are becoming clearer. As the outbreak accelerates, there are early lessons to be learned. The transmissibility of 2019-nCoV—or at least its geographical distribution—seems to be higher and broader than initially expected. Why? Partly this may be because of China's rapid expansion of its transport networks, especially air and high-speed rail. Wuhan is a crucial hub: linking west to Chengdu, south to Guangzhou and Shenzhen, east to Nanjing and Shanghai, and north to Beijing. With much of December a period when the outbreak went unreported and unrecognised, the population exposed to the virus is far greater than first thought—a cause for heightened concern.
“No bats were sold or found at the Huanan seafood market”
Dr. Yuhong writes about The Lancet study by authors Roujian Lu et al., from the China Key Laboratory of Biosafety, National Institute for Viral Disease Control and Prevention, Chinese Center for Disease Control and Prevention, repeating a quote from that paper:
First, the outbreak was first reported in late December 2019, when most bat species in Wuhan are hibernating. Second, no bats were sold or found at the Huanan seafood market, whereas various non-aquatic animals (including mammals) were available for purchase. Third, the sequence identity between 2019-nCoV and its close relatives bat-SL-CoVZC45 and bat-SL-CoVZXC21 was less than 90%. Hence, bat-SL-CoVZC45 and bat-SL-CoVZXC21 are not direct ancestors of 2019-nCoV.
In other words, it isn’t from bats.
That means the entire mainstream media is lying to us about the real origins of the coronavirus.
That same paper goes on to underscore the misinformation in the official explanation, stating, “Many of the initially confirmed 2019-nCoV cases—27 of the first 41 in one report, 26 of 47 in another—were connected to the Wuhan market, but up to 45%, including the earliest handful, were not. This raises the possibility that the initial jump into people happened elsewhere.”
Both Lu (in The Lancet paper linked above) and Lyons-Weiler point to the presence of a SARS binding protein sequence in the coronavirus that allows it to easily infect human cells. As explained in The Epoch Times:
…despite considerable genetics distance between the Wuhan CoV and the human-infecting SARS-CoV, and the overall low homology of the Wuhan CoV S-protein to that of SARS-CoV, the Wuhan CoV S-protein had several patches of sequences in the receptor binding (RBD) domain with a high homology to that of SARS-CoV. The residues at positions 442, 472, 479, 487, and 491 in SARS-CoV S-protein were reported to be at receptor complex interface and considered critical for cross species and human-to-human transmission of SARS-CoV. So to our surprise, despite replacing four out of five important interface amino acid residues, the Wuhan CoV S-protein was found to have a significant binding affinity to human ACE2. …The Wuhan CoV S-protein and SARS-CoV S-protein shared an almost identical 3-D structure in the RBD domain, thus maintaining similar van der Waals and electrostatic properties in the interaction interface. Thus the Wuhan CoV is still able to pose a significant public health risk for human transmission via the S protein–ACE2 binding pathway. (emphasis added)
As Dr. Yuhong asks, “How could this novel virus be so intelligent as to mutate precisely at selected sites while preserving its binding affinity to the human ACE2 receptor? How did the virus change just four amino acids of the S-protein? Did the virus know how to use Clustered Regularly Interspaced Short Palindromic Repeats (CRISPR) to make sure this would happen?”
It couldn’t happen by chance, in other words. The coronavirus is not a random mutation in the wild. It was engineered.
Many other scientists around the world are now investigating the gene sequences found in the coronavirus, and they are increasingly concluding that elements of the virus have been engineered.
Many of those scientists are being threatened and censored. One paper has so far been forced to be withdrawn and revised, no doubt to remove the key conclusions that point to the genetic engineering origins of the coronavirus, but the proof of its engineering cannot be denied forever.
Hope this info is of some use to anyone.
If any of the above Data is wrong, please advise
All the best
Xard777